Abstract #358
Section: Animal Health
Session: Animal Health: Transition cow health
Format: Oral
Day/Time: Tuesday 10:30 AM–10:45 AM
Location: Sebastian I-2
Session: Animal Health: Transition cow health
Format: Oral
Day/Time: Tuesday 10:30 AM–10:45 AM
Location: Sebastian I-2
# 358
Evaluation of recurrence of frequent diseases and disorders in early postpartum dairy cows.
Achilles Vieira-Neto*1, Ana Carolina Parize3, Carlos A. Risco2, José Eduardo P. Santos1, Klibs N. Galvão2, 1Department of Animal Sciences, University of Florida, Gainesville, FL, 2Department of Large Animal Clinical Sciences, University of Florida, Gainesville, FL, 3Escola de Veterinária e Zootecnia, Universidade Federal de Goiás, Goiánia, GO, Brazil.
Key Words: dairy cow, postpartum diseases, disease recurrence
Evaluation of recurrence of frequent diseases and disorders in early postpartum dairy cows.
Achilles Vieira-Neto*1, Ana Carolina Parize3, Carlos A. Risco2, José Eduardo P. Santos1, Klibs N. Galvão2, 1Department of Animal Sciences, University of Florida, Gainesville, FL, 2Department of Large Animal Clinical Sciences, University of Florida, Gainesville, FL, 3Escola de Veterinária e Zootecnia, Universidade Federal de Goiás, Goiánia, GO, Brazil.
Objective was to evaluate if having disease in one lactation (Lac1) would affect the risk of developing disease and calving related problems in the subsequent lactation (Lac2). Holstein cows (n = 1351) from 2 herds in North Florida were used to create 2295 lactation pairs from 4590 lactations occurring between 2006 and 2013. Data were collected within 60 DIM. Data were evaluated using the LOGISTIC procedure of SAS. Having dystocia in Lac1 increased the risk of having it in Lac2 (37.0 vs. 23.7%; P < 0.01). Dystocia in Lac2 was also increased by induced parturition and twins in Lac2. Having twins in Lac1 increased the risk of having it in Lac2 (4.6 vs. 2.4%; P < 0.02). Twins in Lac2 tended (P = 0.09) to be increased in parity > 2 vs. parity 2 in Lact2. Having stillbirth in Lac1 did not affect the risk of having it in Lac2 (5.7 vs. 3.2%; P = 0.12). Stillbirth in Lac2 was increased (P < 0.05) by dystocia and twins in Lac2. Having milk fever in Lac1 increased the risk of having it in Lac2 (38.9 vs. 2.6%; P < 0.01). Milk fever in Lact2 was also increased (P < 0.05) by dystocia and parity ≥ 2 in Lac2, but was decreased by induced parturition in Lac2. Having retained placenta in Lac1 increased the risk of having it in Lac2 (13.1 vs. 6.2%; P = 0.01). Retained placenta in Lact 2 was also increased (P < 0.05) by twins and induced parturition in Lac2. Having metritis in Lac1 increased the risk of having it in Lac2 (20.4 vs. 10.4%; P < 0.02). Metritis in Lact2 was also increased (P < 0.05) by dystocia, induced parturition, twins, stillbirth, retained placenta and ketosis in Lact2. Having ketosis in Lac1 increased the risk of having it in Lac2 (41.9 vs. 17.0%; P = 0.01). Ketosis in Lact2 was also increased (P < 0.05) by induced parturition, metritis, and parity > 2 in Lac2. Having displaced abomasum in Lac1 had a tendency to increase the risk of having it in Lac2 (10.5 vs. 2.1%; P = 0.06). Displaced abomasum in Lact 2 was increased (P < 0.05) by ketosis in Lac2. Having mastitis in Lac1 had a tendency to increase the risk of having it in Lac2 (22.9 vs. 9.3%; P = 0.09). Mastitis in Lact2 was increased (P < 0.05) by parity > 2. In conclusion, with the exception of stillbirth, disease in Lac1 affected the incidence of disease in Lac2.
Key Words: dairy cow, postpartum diseases, disease recurrence
