Abstract #603
Section: Ruminant Nutrition
Session: Ruminant Nutrition: Manipulating rumen function
Format: Oral
Day/Time: Tuesday 2:00 PM–2:15 PM
Location: Panzacola H-2
Session: Ruminant Nutrition: Manipulating rumen function
Format: Oral
Day/Time: Tuesday 2:00 PM–2:15 PM
Location: Panzacola H-2
# 603
The effects of Megalac and a fatty acid prill containing high levels of palmitic acid supplementation on milk fatty acid composition with early lactation dairy cows.
Guiling Ma1, Elliot Block2, Limin Kung3, Joe Harrison*1, C. Merrill2, 1Washington State University, Puyallup, WA, 2Arm & Hammer Animal Nutrition, Princeton, NJ, 3University of Delaware, Newark, DE.
Key Words: milk, fatty acid, dairy
The effects of Megalac and a fatty acid prill containing high levels of palmitic acid supplementation on milk fatty acid composition with early lactation dairy cows.
Guiling Ma1, Elliot Block2, Limin Kung3, Joe Harrison*1, C. Merrill2, 1Washington State University, Puyallup, WA, 2Arm & Hammer Animal Nutrition, Princeton, NJ, 3University of Delaware, Newark, DE.
The objective of this study was to compare 2 fat sources, Megalac (Church & Dwight Co. Inc., Princeton, NJ;) and a high palmitic acid prill (Guarantee-palmitate (min) 80%) on composition of milk fatty acids of early lactation cows in a feeding trial lasting 12 wks. Thirty multiparous Holstein cows were randomly assigned to 1 of 2 diets. Except for the fat products, other feeding ingredients were identical in the diets. Fat products were supplemented at 1.2% (DMI). Milk components were assessed from AM and PM milkings. Milk fatty acids were analyzed with Proc Mixed. The interaction between time and treatment was also analyzed. There were no differences in the composition of short chain fatty acids (C6:0, 8:0, 10:0, 11:0, 12:0, 14:1T, 14:1, 15:0 and 15:1T) between treatments (P > 0.05). Palmitic supplementation increased palmitic acid in milk (37.2 ± 0.5 vs. 40.7 ± 0.6, P < 0.05), while there was a fat source x week interaction (P < 0.05). Megalacsupplementation increased the concentration of stearic acid (10.1 ± 0.3 vs. 8.8 ± 0.3, P < 0.05), and no interaction between fat source and time (P > 0.05). MEGALACTM supplementation also increased (P < 0.05) unsaturated fatty acids C18:1–8T (0.081 ± 0.004 vs. 0.067 ± 0.004), C18:1–9T (0.31 ± 0.03 vs. 0.21 ± 0.03), C18:2 (2.5 ± 0.1 vs.2.2 ± 0.1), C20:1–8-eicosenoate (0.054 ± 0.003 vs. 0.042 ± 0.004), and there was no time interaction (P > 0.05). Megalacsupplementation increased (P < 0.05) C18:1–11T (1.13 ± 0.08 vs.0.89 ± 0.08, P < 0.05), tended to increase C18:1–12C (0.34 ± 0.01 vs. 0.31 ± 0.01, P < 0.07), increased C18:2-cis-9,trans-11 (0.45 ± 0.02 vs. 0.38 ± 0.03, P < 0.06), and there was a time interaction. There was no difference (P > 0.05) between fat sources for C16:1T, C16:1, C17:0, C17:1–10-heptadecenoate, C18:1–10-T, C18:1, C18:1–11-C, C18:3-γ, C18:3-α, C18:2CLA, and C18:2- trans-10,cis-12, in milk. Except for C18:1–12C, there was no difference between AM and PM sampling times (P > 0.05). Our findings suggest that in the future, it is not necessary to separately analyze the AM, PM samples for milk fatty acid analysis. Feeding of Megalacappears to promote the T 11 vs. T 10 pathway of biohydrogenation.
Key Words: milk, fatty acid, dairy
